Redness and Broken Blood Vessels
What is facial redness?
Skin redness refers to visible flushing or persistent pink or red discoloration of the skin. It most commonly appears on the face, particularly across the cheeks, nose, chin, and forehead, although it can occur anywhere on the body. Redness develops when blood vessels in the skin widen, allowing more blood to flow near the surface. This vascular response may be temporary, such as blushing or reacting to heat, or it may become chronic.
Persistent redness is frequently associated with inflammatory skin conditions such as rosacea. In other cases, it may result from sun damage, irritation from skin care products, allergic reactions, eczema, or certain autoimmune disorders. Repeated exposure to ultraviolet radiation can weaken blood vessel walls over time, making redness more noticeable and longer lasting.
Symptoms vary depending on the cause. Some individuals experience only visible redness, while others report burning, stinging, dryness, swelling, or acne-like bumps. When redness involves the eyes, symptoms may include irritation or a gritty sensation. A thorough skin examination helps determine whether redness is purely vascular or part of a broader inflammatory condition.
Diagnosis begins with a detailed history, including triggers such as sun exposure, spicy foods, alcohol, temperature changes, stress, or new skin products. Careful inspection of the skin allows differentiation between diffuse redness, inflammatory lesions, and visible dilated vessels. In most cases, diagnosis is clinical and does not require laboratory testing.
Broken blood vessels, often called spider veins or telangiectasias, are small, dilated capillaries visible just beneath the surface of the skin. They commonly appear on the face, especially around the nose and cheeks, as well as on the legs. These vessels look like thin red, pink, or purple lines that may branch outward in a web-like pattern.
Despite the term “broken,” the vessels are not actually ruptured. Instead, they have become permanently enlarged. Chronic sun exposure is one of the most common causes, as ultraviolet radiation weakens vessel walls and surrounding connective tissue. Rosacea, aging, genetic predisposition, hormonal changes, and repeated facial flushing also contribute to their development.
Diagnosis is straightforward and based on visual examination. The pattern and distribution of vessels help distinguish cosmetic telangiectasias from vascular disorders that affect deeper veins. If spider veins on the legs are extensive or accompanied by swelling or discomfort, additional evaluation may be recommended to rule out underlying venous insufficiency.
Sun exposure remains a primary driver in the development of chronic redness and telangiectasias. Ultraviolet radiation damages collagen and elastin, reducing support for small vessels and making them more apparent.
Genetics also play a significant role. Individuals with fair skin, a family history of rosacea, or a tendency toward flushing are more prone to persistent redness and broken vessels. Hormonal fluctuations during pregnancy or menopause may increase vascular visibility. Trauma to the skin, aggressive exfoliation, and long-term use of topical steroids can further weaken vessel integrity.
Medical management with oral and topical medications is an option for some patients, but for most with persistent redness or broken blood vessels, laser is recommended. Richey Dermatology is proud to offer Vbeam® or pulsed dye laser, the most effective laser for removing spider veins, cherry angiomas, vascular birthmarks and improving redness. Dr. Richey has performed this laser thousands of times and is one of only a small group of physicians who were Harvard fellowship-trained in laser and cosmetic dermatology by Rox Anderson, the inventor of pulsed dye laser.
At a Glance
Dr. Patricia Richey
- Board-certified dermatologist
- Fellowship-trained and board-certified Mohs surgeon
- Harvard fellowship-trained in lasers and cosmetics
- Author of innovative dermatology research and publications
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